David B. Haslam, MD

Director, Antimicrobial Stewardship Program

Clinical Director, Division of Infectious Diseases

Associate Professor, UC Department of Pediatrics

Phone 513-803-1170

Email david.haslam@cchmc.org

Pediatric infectious diseases; host-microbial interactions; innate immunity

MD: University of Calgary, Calgary, Alberta, 1987.

Rotating Internship: McGill University, Montreal, Quebec, 1988.

Residency: University of Ottawa, Ottawa, Quebec, 1991.

Fellowship: Washington University School of Medicine, St. Louis, MO, 2005.

Certification: Pediatric Infectious Diseases, 1997.

El Feghaly R, Stauber J, Tarr PI, Haslam DB. Intestinal inflammatory biomarkers and outcome in pediatric Clostridium difficile infections. J Pediatr. 2013. In press.

El Feghaly R, Stauber J, Tarr PI, Haslam DB. Viral co-infections are common and are associated with higher bacterial burden in children diagnosed with Clostridium difficile colitisJPGN. In press.

El Feghaly R, Stauber J, Deych E, Gonzales C, Tarr PI, Haslam DB. Markers of intestinal Inflammation, not bacterial burden, correlate with clinical outcomes in Clostridium difficile infection. Clin Infect Dis. 2013;56(12):1713-21.

Bobo, LD, El Feghaly R, Chen YS, Dubberke ER, Han Z, Baker A, Li J, Burnham CA, Haslam DB. MAPK-activated protein kinase 2 contributes to Clostridium difficile-associated inflammationInfect Immun. 2013;81(3):713-722.

Chumbler NM, Farrow MA, Lapierre L, Franklin J, Haslam DB, Goldenring J, Lacey B. Clostridium difficile Toxin B causes epithelial cell necrosis through an autoprocessing-independent mechanismPlos Pathogens. 2012;8(12):1-12.

Saenz JB, Li J, Haslam DB. The MAP kinase-activated protein kinase 2 (MK2) contributes to the Shiga toxin-induced inflammatory response. Cell Microbiol. 2010;12:516-29.

Saenz JB, Sun, W, Chang, JW, Li, J, Bursulaya, B., Gray, NS, Haslam, DB. Golgicide A reveals essential roles for GBF1 in Golgi assembly and function. Nature Chem Biol.2009;5(3):157-65.