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Undergraduate institution: University of Massachusetts-Boston, Boston, Massachusetts
After graduating with a major in Biology and a minor in Chemistry, I worked in an Infectious Disease diagnostic laboratory. While I had learned and gained a great deal from the diagnostic field of laboratory science, I sought the challenge and in-depth scientific questions that research had to offer.I was first attracted to the Molecular and Developmental Biology (MDB) program at UC because of its close affiliation with one of the top ranking children’s hospitals in the country. I was interested in a program that not only performed basic but also translational research, and the MDB program at Cincinnati Children’s Hospital Medical Center was an excellent option for both.
Upon arrival in Cincinnati, I was particularly impressed with the brand new, state- of- the- art, research facility. I felt completely at ease with student and faculty members during the interview process. There was a real sense of community within the program. It was clear that interactions between students and faculty were encouraged.
The extensive range of knowledge and research among the faculty provides many opportunities for collaboration, discussions and sharing of resources. It is comforting to know that expertise in any given field is most likely within the same building if needed. Students also benefit from interactions with well-known scientists visiting for weekly seminars.
Cincinnati is a very affordable place to live. The competitive stipend offered by the graduate program allowed for a smooth transition from a working paycheck to a student paycheck. There are numerous, ongoing activities within the city including festivals, sporting events and community service efforts. Life as a graduate student is both manageable and enjoyable here.
My decision to study Molecular and Developmental Biology at Cincinnati Children’s was an easy one. I am constantly learning and being challenged. It is an exciting environment to investigate medically relevant research at an exceptional institution.
Wang IC, Zhang Y, Snyder J, Sutherland MJ, Burhans MS, Shannon JM, Park HJ, Whitsett JA, Kalinichenko VV. Increased expression of FoxM1 transcription factor in respiratory epithelium inhibits lung sacculation and causes Clara cell hyperplasia. Dev Biol. 2010;347(2):301-14. PMCID: 2957513.
Sutherland MJ, Ware SM. Disorders of left-right asymmetry: heterotaxy and situs inversus. Am J Med Genet C Semin Med Genet. 2009;151C(4):307-17.
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