Publications
Xu, M., Guo, Y., Vorhees, C.V., and Zhang, J. (2000). Behavioral responses to cocaine and amphetamine in mice lacking the dopamine D1 receptor. Brain Res., 852, 198-207.
Slane, J.H., Lee, H.S., Vorhees, C.V., Zhang, J., and Xu, M. (2000). DNA fragmentation factor 45 deficient mice exhibit enhanced spatial learning and memory compared to wild-type control mice. Brain Res., 867, 70-79.
Reed, T.M., Repaske, D.R., Snyder, G.L., Greengard, P., and Vorhees, C.V. (2002). Phosphodiesterase 1B knock-out mice exhibit exaggerated locomotor hyperactivity and DARPP-32 phosphorylation in response to dopamine agonists and display impaired spatial learning. J. Neurosci., 22, 5188-5197.
Slane-McQuade, J.M., Lee, H.S., Vorhees, C.V., Xu, M., and Zhang, J. (2002). DNA fragmentation factor 45 knockout mice exhibit longer memory retention in the novel object recognition task compared to wild-type mice. Physiol. Behav., 76, 315-320.
Zhang, J., Slane-McQuade, J.M., Vorhees, C.V. and Xu, M. (2002). Hippocampal expression of c-fos is not essential for spatial learning. Synapse, 46(2), 91-99.
Skelton, M.R., Ponniah, S., Wang, D.Z-M., Doetschman, T., Vorhees, C.V., and Pallen, C.J. (2003). Protein tyrosine phosphatase alpha (PTP) knockout mice show deficits in Morris water maze learning, decreased locomotor activity, and decreases in anxiety. Brain Res., 984, 1-10.
Brunskill, E.W., Ehrman, L.A., Williams, M.T., Klanke, J., Hammer, D., Schaefer, T.L., Sah, R., Dorn, G.W. II, Potter, S.S., and Vorhees, C.V. (2005). Abnormal neurodevelopmental, behavioral, and neurotransmitter signaling in Npas3-deficient mice. Eur. J. Neurosci., 22(6), 1265-1276.
Ehrman, L.A., Williams, M.T., Schaefer, T.L., Gudelsky, G.A., Reed, T.M., Fienberg, A.A., Greengard, P., and Vorhees, C.V. (2006). Phosphodiesterase 1B modulates the effects of methamphetamine on locomotor activity and spatial learning through a DARPP-32-dependent pathway: Evidence from PDE1B-DARPP32 double-knockout mice. Genes Brain Behav., 5, 540-551.
McAuliffe, J.J., Miles, L., and Vorhees, C.V. (2006). Adult neurological function following neonatal hypoxia-ischemia in a mouse model of the term neonate: water maze performance is dependent on separable cognitive and motor components. Brain Res., 1118, 208-221.
Moseley, A.E., Williams, M.T., Schaefer, T.L., Bohanan, C.S., Neumann, J.C., Behbehani, M.M., Vorhees, C.V., and Lingrel, J.B. (2007). Deficiency in Na,K-ATPase α isoform genes alters spatial learning, motor activity and anxiety in mice. J. Neurosci., 27(3), 616-626.
McAuliffe, J.J., Joseph, B., and Vorhees, C.V. (2007). Isoflurane delayed pre-conditioning reduces immediate mortality and improves striatal function in adult mice following neonatal hypoxia-ischemia. Anes. Analges., 104, 1066-1077.
Siuciak, J.A., McCarthy, S.A., Chapin, D.S., Reed, T.M., Vorhees, C.V., and Repaske, D.R. (2007). Behavioral and neurochemical characterization of mice deficient in the phosphodiesterase 1B (PDE1B) enzyme. Neuropharmacology, 53, 113-124.
Lingrel, J.B, Williams, M.T., Vorhees, C.V, and Moseley, A.E. (2007). Na,K-ATPase and the role of the α isoforms. J. Bioenerg. Biomembr., 39, 385-389.
Pan, D., Sciascia, A., II, Vorhees, C.V., and Williams, M.T. (2008). Progression of behavioral deficits during development in a murine model of Hurler syndrome. Brain Res., 1188, 241-253.
McAuliffe, J.J., Joseph, B., Hughes, E., Miles, L., and Vorhees, C.V. (2008). Metallothionein I,II deficient mice do not exhibit significantly worse long-term behavioral outcomes following neonatal hypoxia-ischemia: MT I,II deficient mice have inherent behavioral impairments. Brain Res., 1190, 175-185.
Loepke, A.W., Istaphanous, G.K., Miles, L., Hughes, E.H., McCann, J.C., Kurth, C.D., Williams, M.T., Vorhees, C.V., and Danzer, S.C. (2008). Effects of neonatal anesthesia on neuronal structure and adult neurocognitive function in mice. Anes. Analges., (In press).
Sun, Yl, Jia, L., Williams, M.T., Zamzow, M., Ran, H., Quinn, B., Aronow, B.J., Vorhees, C.V, Witte, D.P, and Grabowski, G.A. (2008). Gene expression profiling and neurobehavioral assessment in prosaposin deficient mice: Molecular alterations precede neuronal deficits. BMC Neurosci., (In press).
Sun, Y., Witte, D.P., Ran, H., Zamzow, M., Barnes, S., Cheng, H., Han, X., Williams, M.T., Skelton, M.R., Vorhees, C.V., and Grabowski, G.A. (2008). Neurological deficits and glycosphingolipid accumulation in Saposin B deficient mice. Human Mol. Genet., 17, 2345-2356.
