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GFP expression in the developing face of a mouse embryo
Perturbations of the primary cilia lead to a dysmorphic forebrain in the developing mouse.
Histological and immunohistochemical analysis of a mouse model of ciliopathies.
Immunohistochemical analysis of a transgenic mouse model of abnormal forebrain development.
Neurons in vitro stained with antibodies to identify stem cells and differentiated cells.
Transgenic mice used to monitor hedgehog pathway activity in one of our ENU mutants with craniofacial abnormalities.
This is a new ENU mutant we call "Dorothy" for the blood clots often seen in the lower limbs of the mutant embryos.
Click image to enlarge.
Cells cultured in vitro and labeled with Bodipy Dye to highlight intracellular lipids.
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An enlarged brain in a mouse mutant generated as part of our studies on the role of primary cilia in neural development.
Elements of the peripheral nervous system highlighted by Cre-LoxP fate mapping.
Primary cilia on cells in vitro.
Vasculature and cell division in the developing mouse cortex.
AP2 Cre activity in the E9.5 mouse embryo.
DAB immunohistochemistry in the mouse cortex.
Vasculature in the embryonic mouse cortex.
Histological analysis of the mouse embryo.
Increased expression of an autophagy marker in vitro (LC3-RFP).
One of our ENU mutant lines showing stunted growth and dysmorphic craniofacial features.
Tbr2 immunohistochemistry in the developing cortex.
Craniofacial and neural phenotypes in our Ttc21b mutants.
A useful transgenic line to monitor developmental signaling in vivo.
Immunohistochemical analysis of our Ttc21b mutants.
Immunohistochemistry of the embryonic mouse brain.
Primary neurons in vitro.
Skeletal preparation highlighting bony (red) and cartilaginous (blue) elements of the perinatal mouse embryo.
A tough PCR genotyping reaction successfully working.
Transfected cells in vitro.
Rolf Stottmann, PhD
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