Sawtell Lab

WEAVER, TE; SARIN, VK; SAWTELL, N; HULL, WM; WHITSETT, JA. IDENTIFICATION OF SURFACTANT PROTEOLIPID-SP-B IN HUMAN SURFACTANT AND FETAL LUNG. Journal of applied physiology (Bethesda, Md. : 1985). 1988; 65:982-987.

Doll, JR; Hoebe, K; Thompson, RL; Sawtell, NM. Resolution of herpes simplex virus reactivation in vivo results in neuronal destruction. PLoS Pathogens. 2020; 16:e1008296-e1008296.

Sawtell, NM; Thompson, RL. HSV Mutant Generation and Dual Detection Methods for Gaining Insight into Latent/Lytic Cycles In Vivo. Methods in molecular biology (Clifton, N.J.). 2020; 2060:219-239.

Thompson, RL; Sawtell, NM. Targeted Promoter Replacement Reveals That Herpes Simplex Virus Type-1 and 2 Specific VP16 Promoters Direct Distinct Rates of Entry Into the Lytic Program in Sensory Neurons in vivo. Frontiers in Microbiology. 2019; 10.

Doll, JR; Thompson, RL; Sawtell, NM. Infectious Herpes Simplex Virus in the Brain Stem Is Correlated with Reactivation in the Trigeminal Ganglia. Journal of Virology. 2019; 93.

Cohen, C; Corpet, A; Roubille, S; Maroui, MA; Poccardi, N; Rousseau, A; Kleijwegt, C; Binda, O; Texier, P; Sawtell, N; et al. Promyelocytic leukemia (PML) nuclear bodies (NBs) induce latent/quiescent HSV-1 genomes chromatinization through a PML NB/Histone H3.3/H3.3 Chaperone Axis. PLoS Pathogens. 2018; 14:e1007313-e1007313.

Pandey, U; Renner, DW; Thompson, RL; Szpara, ML; Sawtell, NM. Inferred father-to-son transmission of herpes simplex virus results in near-perfect preservation of viral genome identity and in vivo phenotypes. Scientific Reports. 2017; 7.

Doll, JR; Sawtell, NM. Analysis of Herpes Simplex Virus Reactivation in Explant Reveals a Method-Dependent Difference in Measured Timing of Reactivation. Journal of Virology. 2017; 91.

Sawtell, NM; Thompson, RL. De Novo Herpes Simplex Virus VP16 Expression Gates a Dynamic Programmatic Transition and Sets the Latent/Lytic Balance during Acute Infection in Trigeminal Ganglia. PLoS Pathogens. 2016; 12:e1005877-e1005877.