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Research Assistant III
I am the research assistant and lab manager for the Lucas lab. Along with assisting in others’ experiments and analyses, my project involves investigating the role of TNFa in bone marrow recovery post-transplant.
I am a graduate student in Cincinnati Children's Hospital Medical Center’s Molecular and Developmental Biology PhD Program. My projects in the Lucas Lab involve remodeling of the bone marrow vascular niche following bone marrow injury and its impact on hematopoiesis.
I am a student in the Immunology Graduate Program working on projects concerning bone marrow remodeling during emergency granulopoiesis and vascular regeneration. Work on one of these projects won the American Association of Immunologists Young Investigator award for best presentation in 2018. Hematology is particularly interesting due to clinical use of bone marrow transplants in treating hematological disorders. While frequently used, its complex development and regulation is vastly understudied.
I am a PhD student in the Immunology Graduate Program at the University of Cincinnati/Cincinnati Children’s Hospital. My graduate training aims to understand the hematopoietic stem cell niche in the developing bone marrow microenvironment.
I obtained my PhD at Peking Union Medical College. After that, I completed a first postdoc in Doug Engel’s lab at the University of Michigan investigating transcriptional regulation in T-cells. My projects in the Lucas lab focus on understanding how TNFa regulates hematopoiesis and how we can map erythroid differentiation in the bone marrow.
I am a postdoctoral fellow in the Lucas lab. I obtained my Doctor of Medicine degree at the Institute of Hematology & Blood Diseases Hospital, Chinese Academy of Medical Science & Peking Union Medical College. My projects focus on mapping myelopoiesis and its regulation in the bone marrow. The goal of our work is to find ways to regulate specific steps of myelopoiesis which can benefit patients with bone marrow failure, myeloid leukemia, MDS, or chemotherapy-induced agranulocytosis.
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