BS: Microbiology and Bioanalysis, University of Antioquia, Colombia, 2006
PhD: Immunobiology, University of Cincinnati, Cincinnati, OH, 2014
PostDoc: Division of Immunobiology, Cincinnati Children’s Hospital Medical Center, Cincinnati, OH, 2022
Obesity; T2D; NAFLD
Immune regulation; inflammation; obesity; diabetes; liver disease; aging
Conditional overexpression of PPARα in intestinal epithelium diminishes GIP enteroendocrine cells and circulating hormone levels. Molecular Metabolism. 2026; 112:102429.
Glutamine metabolism as a rheostat of CD4+ T cell pathogenic function in metabolic dysfunction-associated steatotic liver disease. Cell Metabolism. 2026.
TRANCE is a unique marker of chronic pancreatitis in children. JCI Insight. 2026; 11(16).
B cells just got a workout. Cell. 2026; 189(11):3181-3183.
Ambient temperature regulates CD4+ T cell tonic T cell receptor signaling and responsiveness. iScience. 2026; 29(5):115801.
Distinct inflammatory programming of thoracic cavity white adipose immune cells regulates influenza pathogenesis. Journal of Infectious Diseases. 2026.
Patient-derived colon epithelial organoids reveal lipid-related metabolic dysfunction in pediatric ulcerative colitis. Nature Communications. 2025; 16(1):11026.
Maternal metabolic dysfunction-associated steatotic liver disease during pregnancy and its impact on offspring steatosis. Clinical Nutrition ESPEN. 2025; 70:327-335.
A shift in PKM2 oligomeric state instructs adipocyte inflammatory potential. JCI Insight. 2025; 10(22).
990: DESCRIBING DISTINCT IMMUNOPROTEIN PATTERNS WITHIN PEDIATRIC PATIENTS FROM INITIAL ACUTE PANCREATITIS ATTACK THROUGH CHRONIC PANCREATITIS USING NULISA PROTEOMIC ASSAY REVEALS AN INCREASED EXPRESSION OF TRANCE. Gastroenterology. 2025; 169(1):s-230-s-231.