Katherine A. Bowers, PhD, MPH
- Member, Division of Biostatistics and Epidemiology
- Professor, UC Department of Pediatrics
About
Biography
My interest in research came from a long-standing desire to understand the etiology of autism spectrum disorders. After joining Cincinnati Children’s in 2012, I began to study child development more broadly. My research is primarily in perinatal epidemiology, and my specific areas of interest include exposures and conditions during pregnancy and their implications for offspring health.
Two of my primary projects focus on psychosocial factors and diabetes experienced in pregnancy. The first one is about how adversity experienced by individuals in poverty affects child development and how DNA methylation may underlie this effect. The second project follows the offspring of women who had diabetes in pregnancy. I am interested in the long-term effects of metabolic and neurobehavioral impairments.
PhD: Johns Hopkins Bloomberg School of Public Health, Baltimore, MD.
Post-doctoral Fellowship: National Institute of Child Health and Human Development, NIH, Bethesda MD.
Interests
Infant and child development; epigenetics; pregnancy; health disparities
Research Areas
Biostatistics and Epidemiology
Publications
Self-puberty staging in endocrine encounters during the COVID pandemic. Frontiers in Endocrinology. 2024; 15:1487329.
Community-level and maternal individual-level social support and offspring epigenetic aging. Annals of Epidemiology. 2024; 97:144.
Father involvement during pregnancy and maternal and neonatal health outcomes. Annals of Epidemiology. 2024; 97:145.
The association between everyday discrimination and perinatal depression in a birth cohort in Cincinnati, Ohio. Annals of Epidemiology. 2024; 97:143.
Association between maternal prenatal depressive symptoms and offspring epigenetic aging at 3-5 weeks. Annals of Epidemiology. 2024; 93:1-6.
Current Recommended Estrogen Dosing for Pubertal Induction in Turner Syndrome Results in Normal Uterine Growth. The Journal of Clinical Endocrinology and Metabolism. 2024; 109:e1040-e1047.
Neonatal AVPR1a Methylation and In-Utero Exposure to Maternal Smoking. Toxics. 2023; 11:855.
THU203 Self-puberty Staging In Endocrine Encounters During The COVID Pandemic. Journal of the Endocrine Society. 2023; 7:bvad114.1454.
1149-P: Health Literacy and Numeracy Based on The Newest Vital Sign Is Not Associated with Hemoglobin A1c in Caretakers or Youth with Type 1 Diabetes. Diabetes. 2023; 72.
Epigenome-wide association of neonatal methylation and trimester-specific prenatal PM2.5 exposure. Environmental Epidemiology. 2022; 6:e227.
From the Blog
DNA Methylation May Serve as Biomarker for Maternal Stress Passed to Children
Katherine A. Bowers, PhD, MPH, Alonzo T. Folger, PhD, MS3/21/2022
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