Heart disease in a mutant mouse model of spontaneous eosinophilic myocarditis maps to three loci.
Zimmermann, N; Jr, GW J; Homan, SM; Prows, DR.
BMC Genomics.
2019;
20.
Gpr63 is a modifier of microcephaly in Ttc21b mouse mutants.
Snedeker, J; Jr, GW J; Paulding, DF; Abdelhamed, Z; Prows, DR; Stottmann, RW.
PLoS Genetics.
2019;
15:e1008467-e1008467.
Characterization of a mouse model of hypereosinophilia-associated heart disease.
Prows, DR; Klingler, A; Jr, GW J; Homan, SM; Zimmermann, N.
American Journal of Physiology: Heart and Circulatory Physiology.
2019;
317:H405-H414.
Genetic susceptibility to toxicologic lung responses among inbred mouse strains following exposure to carbon nanotubes.and profiling of underlying gene networks.
Frank, EA; Carreira, VS; Shanmukhappa, K; Medvedovic, M; Prows, DR; Yadav, JS.
Toxicology and Applied Pharmacology.
2017;
327:59-70.
Stem Cell-Specific Mechanisms Ensure Genomic Fidelity within HSCs and upon Aging of HSCs.
Moehrle, BM; Nattamai, K; Brown, A; Florian, MC; Ryan, M; Vogel, M; Bliederhaeuser, C; Soller, K; Prows, DR; Abdollahi, A; et al.
Cell Reports.
2015;
13:2412-2424.