A photo of Jerzy Stanek.

Jerzy W. Stanek, MD, PhD

  • Staff Pathologist, Division of Pathology
  • Director, Placental & Perinatal Pathology
  • Professor, UC Department of Pathology and Laboratory Medicine
  • UC Department of Pediatrics



Intrauterine hypoxia takes place when the uterus does not have an adequate supply of oxygen. Once this occurs, intrauterine growth restriction could occur and damage the brain and spinal cord. This condition may lead to a higher mortality rate, including sudden infant death syndrome (SIDS). As such, it is essential to pursue scientific research into in utero-fetal hypoxia and other perinatal complications.

My primary research interests are in placental and perinatal pathology. In particular, I want to refine the placental diagnostic criteria of in utero-fetal hypoxia and perinatal complications.

At the beginning of my career, I was an obstetrician, gynecologist, reproductive endocrinologist and clinical perinatologist in Poland. In 1994, I came to Cincinnati Children’s as a perinatal and placental pathologist.

In the field of gynecology, I developed a new approach for operative treatment of female urinary incontinence. In the discipline of placental pathology, I described multiple placental hypoxia-related lesions, including:

  • Chorionic microcysts of placental membranes and chorionic disc
  • Increased amount of extravillous trophoblasts, occult placenta accreta and perivascular stem edema
  • Laminar necrosis of placental membranes

My recognitions include receiving five residency teaching awards from the University of Cincinnati and an award from the President of the Washington State Society of Pathologists.

I am an author of more than 100 peer-reviewed publications and 100 abstracts. In addition, I am a co-author of three academic textbooks on clinical perinatology. Some respected journals that have published my studies include Pediatric and Developmental Pathology: The Official Journal of the Society for Pediatric Pathology and the Paediatric Pathology Society, Placenta, Journal of Medical Primatology and Archives of Pathology, and Laboratory Medicine.

Additional Languages



Cesarean section per se is not a risk factor for non-anatomical placenta creta. Stanek, J. Journal of the International Federation of Gynaecology and Obstetrics. 2022.

Umbilical cord compromise versus other clinical conditions predisposing to placental fetal vascular malperfusion. Stanek, J. Placenta. 2022; 127:8-11.

Distal villous lesions are clinically more relevant than proximal large muscular vessel lesions of placental fetal vascular malperfusion. Stanek, J. Histology and Histopathology. 2022; 37:365-372.

Placental CD34 immunohistochemistry in fetal vascular malperfusion in stillbirth. Stanek, J; Drach, A. Journal of Obstetrics and Gynaecology Research. 2022; 48:719-728.

Shallow Placentation: A Distinct Category of Placental Lesions. Stanek, J. American Journal of Perinatology. 2021.

Temporal heterogeneity of placental segmental fetal vascular malperfusion: timing but not etiopathogenesis. Stanek, J. Virchows Archiv. 2021; 478:905-914.

CD34 immunostain increases sensitivity of the diagnosis of fetal vascular malperfusion in placentas from ex-utero intrapartum treatment. Stanek, J. Journal of Perinatal Medicine. 2021; 49:203-208.

Grading placental fetal vascular malperfusion and short-term perinatal outcome. Stanek, J. Polish Journal of Pathology. 2021; 71:291-300.

Lymphocytic Colitis With Increased Apoptosis: A Marker of Mutation in T-Cell-Mediated Immunity?. Bernieh, A; Hakar, M; Stanek, J. Pediatric and Developmental Pathology. 2020; 23:443-447.

Placenta Creta: A Spectrum of Lesions Associated with Shallow Placental Implantation. Stanek, J. Obstetrics and Gynecology International. 2020; 2020.

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