A photo of Jocelyn Biagini Myers, PhD.

Assistant Professor, UC Department of Pediatrics

513-803-1110

Biography & Affiliation

Biography

Dr. Biagini Myers is a molecular epidemiologist who began her training with a master’s degree in epidemiology 2004 and subsequent doctorate in molecular epidemiology at the University of Cincinnati (UC) in 2008. She was also a fellow in the Molecular Epidemiology in Children’s Environmental Health training program at UC from 2001-2004. During her time at UC, she was an integral part of building a new birth cohort of children at high risk for developing allergic disease and her research interests in secondhand smoke exposures and allergic disease pathogenesis were established. Her dissertation work evaluated genetic variation in nicotine metabolism-related genes and their effects on hair cotinine levels and wheezing in young children.

Following her successful dissertation defense in 2008, she began a postdoctoral fellowship at Cincinnati Children's. From 2008-2011, she collaborated on a project focused on characterizing epithelial genes in allergic diseases and delineating the mechanisms by which they contribute to the allergic response. She also assisted collaborators in developing a custom SNP-chip for studying genes related to asthma and atopic dermatitis. In addition, she was successful in receiving funding in 2009 with a PI at UC to evaluate the contributions of genetic variation in nicotine metabolism and oxidative stress-related genes in pediatric asthma development.

Dr. Biagini Myers’ long-term goal is to develop her research career as a molecular epidemiologist focused on reducing and preventing secondhand smoke-related pediatric asthma and allergic disease. She is also interested in intervention-based research for early-life prevention and reduction of smoke exposures in children and providing further evidence to promote public policies for smoking bans around children.

Academic Affiliation

Assistant Professor, UC Department of Pediatrics

Department

Asthma

Science Blog

Compromised Skin Outweighs Inheritance as Allergy Risk

Compromised Skin Outweighs Inheritance as Allergy Risk

Jocelyn M. Biagini Myers, PhD2/18/2020

Education

BS: Xavier University, Cincinnati, OH, 2001.

MS: University of Cincinnati, Cincinnati, OH, 2004.

PhD: University of Cincinnati, Cincinnati, OH, 2008.

Postdoctoral Fellowship: Division of Asthma Research, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, 2008-2011.

Publications

Selected Publication

A Pediatric Asthma Risk Score to better predict asthma development in young children. Myers, JM B; Schauberger, E; He, H; Martin, LJ; Kroner, J; Hill, GM; Ryan, PH; LeMasters, GK; Bernstein, DI; Lockey, JE; et al. Journal of Allergy and Clinical Immunology. 2019; 143:1803-1810.e2.

Age and early maternal smoking contribute to epithelial cell IL-13 responsiveness in a pediatric asthma population. McAlees, JW; Baker, T; Kaur, D; McKnight, C; Lindsley, A; Strait, RT; Zhang, X; Myers, JM B; Kovacic, MB; Lewkowich, IP. Allergy: European Journal of Allergy and Clinical Immunology. 2019; 74:2485-2487.

The pediatric asthma risk score (PARS): making the move to the most accurate pediatric asthma risk screening tool. Sherenian, MG; Biagini Myers, JM; Martin, LJ; Khurana Hershey, GK. Expert Review of Clinical Immunology. 2019; 15:1115-1118.

Vitamin D supplementation attenuates asthma development following traffic-related particulate matter exposure. Bolcas, PE; Brandt, EB; Zhang, Z; Biagini Myers, JM; Ruff, BP; Khurana Hershey, GK. Journal of Allergy and Clinical Immunology. 2019; 143:386-394.e3.

The antiprotease SPINK7 serves as an inhibitory checkpoint for esophageal epithelial inflammatory responses. Azouz, NP; Ynga-Durand, MA; Caldwell, JM; Jain, A; Rochman, M; Fischesser, DM; Ray, LM; Bedard, MC; Mingler, MK; Forney, C; et al. Science Translational Medicine. 2018; 10:eaap9736-eaap9736.

Ohio Pediatric Asthma Repository: Opportunities to Revise Care Practices to Decrease Time to Physiologic Readiness for Discharge. Simmons, JM; Biagini Myers, JM; Martin, LJ; Kercsmar, CM; Schuler, CL; Pilipenko, VV; Kroner, JW; He, H; Austin, SR; Nguyen, H; et al. Hospital Pediatrics. 2018; 8:305-313.

Eosinophilic esophagitis (EoE) genetic susceptibility is mediated by synergistic interactions between EoE-specific and general atopic disease loci. Martin, LJ; He, H; Collins, MH; Abonia, JP; Myers, JM B; Eby, M; Johansson, H; Kottyan, LC; Hershey, GK K; Rothenberg, ME. Journal of Allergy and Clinical Immunology. 2018; 141:1690-1698.

High number of early respiratory infections in association with allergic sensitization to mold promotes childhood asthma. Ramirez, LP; Wendroth, H; Martin, LJ; Pilipenko, VV; He, H; Kroner, J; Ryan, PH; LeMasters, GK; Lockey, JE; Bernstein, DI; et al. Journal of Allergy and Clinical Immunology. 2018; 141:1921-1924.e4.

Nasal DNA methylation is associated with childhood asthma. Zhang, X; Myers, JM B; Burleson, JD; Ulm, A; Bryan, KS; Chen, X; Weirauch, MT; Baker, TA; Kovacic, MS B; Ji, H. Epigenomics. 2018; 10:629-641.

Adjunctive Pharmacotherapies in Children With Asthma Exacerbations Requiring Continuous Albuterol Therapy: Findings From The Ohio Pediatric Asthma Repository. Shein, SL; Farhan, O; Morris, N; Mahmood, N; Alter, SJ; Biagini Myers, JM; Gunkelman, SM; Kercsmar, CM; Khurana Hershey, GK; Martin, LJ; et al. Hospital Pediatrics. 2018; 8:89-95.