A photo of J. Pablo Abonia.

J. Pablo Abonia, MD

  • Associate Professor, UC Department of Pediatrics
When I care for my patients, I always put them first, with the goal of identifying their problems and working to resolve them.

About

Biography

I’m an allergist who specializes in treating children with eosinophilic gastrointestinal disorders, eosinophilic esophagitis, eosinophilia, mast cell disorders and urticaria. My research focuses on finding the most effective therapies for patients with eosinophilic and mast cell disorders.

Seeing my father work as a pediatrician inspired me to follow in his footsteps and choose medicine as a profession. I completed my formal pediatric training at Children’s Hospital of Buffalo and my allergy and immunology training at Brigham and Women’s Hospital in Boston. These experiences provided the education, skills and resources that prepared me to help my patients as a member of the Cincinnati Center for Eosinophilic Disorders (CCED).

When I care for my patients, I always put them first, with the goal of identifying their problems and working to resolve them. I strive to improve control of their symptoms. In my research, I work toward long-term control of diseases related to eosinophilic disorders and mast cell disease. I’ve published a number of journal articles in my field, and I am involved in multicenter clinical trials for eosinophilic disorders.

I was previously the interim director of the Registry for Eosinophilic Disorders. I presented the Joseph E. Ghory, MD, Memorial Lectureship for the Ohio Society of Allergy, Asthma, and Immunology; the 15th Annual Dr. John M. Karibo Memorial Lectureship; and at other national meetings related to eosinophilic and mast cell disorders. I’m also proud to be named one of Cincinnati Magazine’s Top Doctors.

Outside of the hospital, I enjoy spending time with my wife and family.

MD: University of Buffalo, Buffalo, NY, 1997.

Residency: Pediatrics, Children's Hospital of Buffalo, Buffalo, NY, 2000.

Certification: American Board of Pediatrics, 2001; American Board of Allergy and Immunology, 2003.

Interests

Immunodeficiency

Services and Specialties

Allergy and Immunology, Eosinophilic Disorders

Interests

Mast cell progenitor homing / recruitment and its involvement in allergic disorders

Research Areas

Allergy and Immunology, Eosinophilic Disorders

Additional Languages

Spanish

Insurance Information

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Publications

Targeted IL-4Rα blockade ameliorates refractory allergic eosinophilic inflammation in a patient with dysregulated TGF-β signaling due to ERBIN deficiency. Droghini, HR; Abonia, JP; Collins, MH; Milner, JD; Lyons, JJ; Freeman, AF; Mukkada, VA; Risma, KA; Rothenberg, ME; Schwartz, JT. Journal of Allergy and Clinical Immunology: In Practice. 2022; 10:1903-1906.

Single-cell RNA sequencing of mast cells in eosinophilic esophagitis reveals heterogeneity, local proliferation, and activation that persists in remission. Ben-Baruch Morgenstern, N; Ballaban, AY; Wen, T; Shoda, T; Caldwell, JM; Kliewer, K; Felton, JM; Abonia, JP; Mukkada, VA; Putnam, PE; et al. Journal of Allergy and Clinical Immunology. 2022; 149:2062-2077.

Evaluating Eosinophilic Colitis as a Unique Disease Using Colonic Molecular Profiles: A Multi-Site Study. Shoda, T; Collins, MH; Rochman, M; Wen, T; Caldwell, JM; Mack, LE; Osswald, GA; Besse, JA; Haberman, Y; Aceves, SS; et al. Gastroenterology. 2022; 162:1635-1649.

Long-Lasting Dissociation of Esophageal Eosinophilia and Symptoms After Dilation in Adults With Eosinophilic Esophagitis. Safroneeva, E; Pan, Z; King, E; Martin, LJ; Collins, MH; Yang, GY; Capocelli, KE; Arva, NC; Abonia, JP; Atkins, D; et al. Clinical Gastroenterology and Hepatology. 2022; 20:766-775.e4.

Acquired Esophageal Strictures in Children: Morphometric and Immunohistochemical Analyses. Collins, MH; Alexander, ES; Martin, LJ; Grotjan, TM; Mukkada, VA; Sheil, A; Abonia, JP; Putnam, PE; Rothenberg, ME. Pediatric and Developmental Pathology. 2022; 25:124-133.

Prospective Endoscopic Activity Assessment for Eosinophilic Gastritis in a Multisite Cohort. Hirano, I; Collins, MH; King, E; Sun, Q; Chehade, M; Abonia, JP; Bonis, PA; Capocelli, KE; Dellon, ES; Falk, GW; et al. American Journal of Gastroenterology. 2022; 117:413-423.

International Consensus Recommendations for Eosinophilic Gastrointestinal Disease Nomenclature. Dellon, ES; Gonsalves, N; Abonia, JP; Alexander, JA; Arva, NC; Atkins, D; Attwood, SE; Auth, MK H; Bailey, DD; Biederman, L; et al. Clinical Gastroenterology and Hepatology. 2022.

Loss of Endothelial TSPAN12 Promotes Fibrostenotic Eosinophilic Esophagitis via Endothelial Cell-Fibroblast Crosstalk. Shoda, T; Wen, T; Caldwell, JM; Ben-Baruch Morgenstern, N; Osswald, GA; Rochman, M; Mack, LE; Felton, JM; Abonia, JP; Arva, NC; et al. Gastroenterology. 2022; 162:439-453.

Desmoplakin and periplakin genetically and functionally contribute to eosinophilic esophagitis. Shoda, T; Kaufman, KM; Wen, T; Caldwell, JM; Osswald, GA; Purnima, P; Zimmermann, N; Collins, MH; Rehn, K; Foote, H; et al. Nature Communications. 2021; 12.

Developing a standardized approach for assessing mast cells and eosinophils on tissue biopsies: A Work Group Report of the AAAAI Allergic Skin Diseases Committee. Zimmermann, N; Abonia, JP; Dreskin, SC; Akin, C; Bolton, S; Happel, CS; Geller, M; Larenas-Linnemann, D; Nanda, A; Peterson, K; et al. Journal of Allergy and Clinical Immunology. 2021; 148:964-983.

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