Craig A. Erickson, MD
- Associate Professor, UC Department of Psychiatry and Behavioral Neuroscience
- Board Certified
About
Biography
Dr. Erickson has worked to obtain continuous federal, foundation, internal, and industry funding supporting his and his collaborators' research over the last 10 ten years of his career. He is the inventor or co-inventor on many patents focused on translational treatment development in neurodevelopmental disorders that are held at Cincinnati Children's Hospital Medical Center and at his previous employer the Indiana University School of Medicine. He is considered an international expert in the clinical treatment of fragile X syndrome and has similar expertise in fragile X-specific clinical trial development. Dr. Erickson is additionally an avid teacher of future generations of child psychiatrists has received several teaching awards for his work in physician education. He also enjoys mentoring junior faculty in the behavioral and developmental neuropsychiatry sub-field of child psychiatry.
Specifically in research, he and his colleagues have moved forward several repurposed molecules for study in fragile X syndrome and autism spectrum disorder including work with acamprosate, riluzole, ketamine, D cycloserine, and N acetyl cysteine among other repurposed molecules. He also is working now to move several novel molecules into autism and fragile X-specific study using proprietary compounds abandoned from initially intended use that may hold promise in the disorders which he and his colleagues study.
MD: University of Cincinnati College of Medicine, Cincinnati, OH, 2003.
MS: Indiana University School of Medicine, Indianapolis, IN, 2008.
Residency and Fellowship: Indiana University School of Medicine, Indianapolis, IN, 2003-2008.
Certification: Child and Adolescent Psychiatry, 2009; Adult Psychiatry, 2008.
Interests
Fragile X syndrome; inpatient neurodevelopmental disorders acute crisis stabilization
Interests
Fragile X syndrome; autism spectrum disorders; neurodevelopmental disorders; molecular blood markers; quantitative measurement of pathophysiology in developmental disabilities; translational treatment development; psychiatric services for those with developmental disabilities and severe behavior
Publications
ZMYND11 functions in bimodal regulation of latent genes and brain-like splicing to safeguard corticogenesis. Nature Communications. 2025; 16:9010.
Divergent aperiodic slope and alpha dynamics expose cortical excitability gradients in fragile X syndrome. Molecular Autism. 2025; 16:49.
Cognitive Flexibility in Autism: New Tools for Understanding and Treatment. Journal of the American Academy of Child and Adolescent Psychiatry. 2025; 64:s335.
Emerging role of complement system in the induction of neuroinflammation in adenylosuccinate lyase deficiency disorder. Brain, Behavior, and Immunity - Health. 2025; 48:101091.
Beyond the Fragile X protein: neighborhood characteristics explain individual differences in IQ and adaptive behaviors of Fragile X syndrome. Frontiers in Psychiatry. 2025; 16:1636987.
FX ENTRAIN: scientific context, study design, and biomarker driven brain-computer interfaces in neurodevelopmental conditions. Frontiers in Neuroscience. 2025; 19:1618804.
Brief Report: A Double-Blind, Placebo-Controlled, Crossover, Proof-of-Concept Study of Minocycline in Autism Spectrum Disorder. Journal of Autism and Developmental Disorders. 2025; 55:3387-3394.
The Aberrant Behavior Checklist for Fragile X Syndrome: A Qualitative Clinician Evaluation of Content Validity. Journal of Child and Adolescent Psychopharmacology. 2025; 35:399-415.
Alpha-2 Agonists in Autism Spectrum Disorder. Lifespan Treatment for Autistic Individuals. : Oxford University Press (OUP); Oxford University Press (OUP); 2025.
Hyper-extralemniscal model of Fragile X syndrome. Cerebral Cortex. 2025; 35:bhaf141.
From the Blog
Positive Clinical Trial Results for a New Fragile X Treatment
Craig A. Erickson, MD2/11/2025
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