A photo of Halley Wasserman.

Member, Division of Endocrinology

Assistant Professor, UC Department of Pediatrics

513-636-4744

Board Certified

My Biography & Research

Biography

Halley M. Wasserman, MD, is a board-certified pediatric endocrinologist and clinical densitometrist. She heads the Metabolic Bone Clinic at Cincinnati Children's Hospital Medical Center providing clinical care to children and adolescents with various bone and mineral disorders. Her research focuses on identifying risk factors for low bone density and fragility fractures in pediatric patients as childhood and adolescence are critical times for building strong bones and establishing the foundation for skeletal health into adulthood. Currently, her main interests focus on the interaction of hormones and the musculoskeletal system during childhood and identifying novel risk factors for fracture in children with chronic medical conditions. Her long-term goal is to develop and implement targeted interventions for children at high risk for skeletal fragility to decrease the prevalence of fractures.

Clinical Interests

Pediatric endocrinology; calcium and phosphorus mineralization disorders; osteoporosis; pubertal disorders; skeletal growth

Research Interests

Osteoporosis specifically in populations with limited mobility; puberty and reproductive health

Academic Affiliation

Assistant Professor, UC Department of Pediatrics

Departments

Endocrinology, Endocrinology

My Locations

My Education

MD: Northeastern Ohio Medical University, Rootstown, OH, 2009.

Residency: Pediatrics, Cincinnati Children’s Hospital Medical Center, Cincinnati, OH, 2012.

Endocrine Fellowship: Endocrinology, Cincinnati Children's Hospital Medical Center, Cincinnati, OH, 2015.

Research Fellowship: Cincinnati Children's Hospital Medical Center, Cincinnati, OH, 2016.

MS: Clinical and Translational Research, University of Cincinnati, Cincinnati, OH, 2016.

Certification: Pediatrics, 2012; Pediatric Endocrinology, 2015.

My Publications

Outcomes following intravenous bisphosphonate infusion in pediatric patients: A 7-year retrospective chart review. Nasomyont, N; Hornung, LN; Gordon, CM; Wasserman, H. Bone. 2019; 121:60-67.

Outcome of Isolated Absent Septum Pellucidum Diagnosed by Fetal Magnetic Resonance Imaging (MRI) Scan. Vawter-Lee, MM; Wasserman, H; Thomas, CW; Nichols, B; Nagaraj, UD; Schapiro, M; Venkatesan, C. Journal of Child Neurology. 2018; 33:693-699.

Bone fragility in Turner syndrome: Fracture prevalence and risk factors determined by a national patient survey. Wasserman, H; Backeljauw, PF; Khoury, JC; Kalkwarf, HJ; Gordon, CM. Clinical Endocrinology. 2018; 89:46-55.

Pharmacokinetics of IGF-1 in PAPP-A2-Deficient Patients, Growth Response, and Effects on Glucose and Bone Density. Cabrera-Salcedo, C; Mizuno, T; Tyzinski, L; Andrew, M; Vinks, AA; Frystyk, J; Wasserman, H; Gordon, CM; Hwa, V; Backeljauw, P; et al. Journal of Clinical Endocrinology and Metabolism. 2017; 102:4568-4577.

Use of dual energy X-ray absorptiometry in pediatric patients. Wasserman, H; O'Donnell, JM; Gordon, CM. Bone. 2017; 104:84-90.

Bone Mineralization and Fracture Risk Assessment in the Pediatric Population. Wasserman, H; Gordon, CM. Journal of Clinical Densitometry. 2017; 20:389-396.

Unexpected widespread hypophosphatemia and bone disease associated with elemental formula use in infants and children. Ballesteros, LF G; Ma, NS; Gordon, RJ; Ward, L; Backeljauw, P; Wasserman, H; Weber, DR; DiMeglio, LA; Gagne, J; Stein, R; et al. Bone. 2017; 97:287-292.

Low bone mineral density and fractures are highly prevalent in pediatric patients with spinal muscular atrophy regardless of disease severity. Wasserman, HM; Hornung, LN; Stenger, PJ; Rutter, MM; Wong, BL; Rybalsky, I; Khoury, JC; Kalkwarf, HJ. Neuromuscular Disorders. 2017; 27:331-337.

Bilateral cataracts in a 6-yr-old with new onset diabetes: a novel presentation of a known INS gene mutation. Wasserman, H; Hufnagel, RB; Utz, VM; Zhang, K; Valencia, CA; Leslie, ND; Crimmins, NA. Pediatric Diabetes. 2016; 17:535-539.

Two Case Reports of FGF23-Induced Hypophosphatemia in Childhood Biliary Atresia. Wasserman, H; Ikomi, C; Hafberg, ET; Miethke, AG; Bove, KE; Backeljauw, PF. Pediatrics. 2016; 138:e20154453-e20154453.