A photo of Charles Quinn.

Charles T. Quinn, MD, MS

  • Medical Director, Pediatric Sickle Cell Program
  • Director, Erythrocyte Diagnostic Laboratory
  • Professor, UC Department of Pediatrics



Dr. Quinn conducts patient-oriented and translational research in sickle cell disease and thalassemia. He is the medical director of the Pediatric Sickle Cell Disease Program and the medical director of the Erythrocyte Diagnostic Laboratory at Cincinnati Children’s Hospital. He is also the medical director of the Ohio Department of Health Regional Sickle Cell Services Program – Region 1.

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Early initiation of hydroxyurea (hydroxycarbamide) using individualised, pharmacokinetics-guided dosing can produce sustained and nearly pancellular expression of fetal haemoglobin in children with sickle cell anaemia. Quinn, CT; Niss, O; Dong, M; Pfeiffer, A; Korpik, J; Reynaud, M; Bonar, H; Kalfa, TA; Smart, LR; Malik, P; et al. British Journal of Haematology. 2021; 194:617-625.


Left atrial dysfunction in sickle cell anemia is associated with diffuse myocardial fibrosis, increased right ventricular pressure and reduced exercise capacity. Alsaied, T; Niss, O; Tretter, JT; Powell, AW; Chin, C; Fleck, RJ; Cnota, JF; Malik, P; Quinn, CT; Nagueh, SM; et al. Scientific Reports. 2020; 10.


Diastolic dysfunction is associated with exercise impairment in patients with sickle cell anemia. Alsaied, T; Niss, O; Powell, AW; Fleck, RJ; Cnota, JF; Chin, C; Malik, P; Quinn, CT; Taylor, MD. Pediatric Blood and Cancer. 2018; 65.


Association between diffuse myocardial fibrosis and diastolic dysfunction in sickle cell anemia. Niss, O; Fleck, R; Makue, F; Alsaied, T; Desai, P; Towbin, JA; Malik, P; Taylor, MD; Quinn, CT. Blood. 2017; 130:205-213.


Biochemical surrogate markers of hemolysis do not correlate with directly measured erythrocyte survival in sickle cell anemia. Quinn, CT; Smith, EP; Arbabi, S; Khera, PK; Lindsell, CJ; Niss, O; Joiner, CH; Franco, RS; Cohen, RM. American Journal of Hematology. 2016; 91:1195-1201.


Sickle cell anemia mice develop a unique cardiomyopathy with restrictive physiology. Bakeer, N; James, J; Roy, S; Wansapura, J; Shanmukhappa, SK; Lorenz, JN; Osinska, H; Backer, K; Huby, A; Shrestha, A; et al. Proceedings of the National Academy of Sciences of the United States of America. 2016; 113:E5182-E5191.


Cardiomyopathy With Restrictive Physiology in Sickle Cell Disease. Niss, O; Quinn, CT; Lane, A; Daily, J; Khoury, PR; Bakeer, N; Kimball, TR; Towbin, JA; Malik, P; Taylor, MD. JACC: Cardiovascular Imaging. 2016; 9:243-252.

Consensus definition of essential, optimal, and suggested components of a pediatric sickle cell disease center. Hulbert, ML; Manwani, D; Meier, ER; Alvarez, OA; Brown, RC; Callaghan, MU; Campbell, AD; Coates, TD; Frei-Jones, MJ; Hankins, JS; et al. Pediatric Blood and Cancer. 2022.

New therapeutics for children with sickle cell disease: A time for celebration, caution, or both?. Quinn, CT; Ware, RE. Pediatric Blood and Cancer. 2022; 69.

An Immersive Virtual Reality Curriculum for Pediatric Hematology Clinicians on Shared Decision-making for Hydroxyurea in Sickle Cell Anemia. Real, FJ; Hood, AM; Davis, D; Cruse, B; Klein, M; Johnson, Y; McTate, E; Brinkman, WB; Hackworth, R; Hackworth, K; et al. Journal of Pediatric Hematology/Oncology. 2022; 44:e799-e803.

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