Charles T. Quinn, MD, MS
- Medical Director, Pediatric Sickle Cell Program
- Director, Erythrocyte Diagnostic Laboratory
- Professor, UC Department of Pediatrics
- charles.quinn@cchmc.org
- Board Certified
About
Biography
Dr. Quinn conducts patient-oriented and translational research in sickle cell disease and thalassemia. He is the medical director of the Pediatric Sickle Cell Disease Program and the medical director of the Erythrocyte Diagnostic Laboratory at Cincinnati Children’s Hospital. He is also the medical director of the Ohio Department of Health Regional Sickle Cell Services Program – Region 1.
MD: University of Texas Southwestern Medical Center, Dallas, TX, 1994.
MS: University of Texas Southwestern Medical Center, Dallas, TX, 2008.
Residency & Chief Residency: Children's Medical Center Dallas, Dallas, TX; University of Texas Southwestern Medical Center, Dallas, TX, 1998.
Fellowship: Children's Medical Center Dallas, Dallas, TX; University of Texas Southwestern Medical Center, Dallas, TX, 2001.
Certifications: Pediatrics, 1998; Pediatric Hematology-Oncology, 2002.
Interests
Sickle cell disease; thalassemia; hemoglobinopathies; genetics of hemoglobinopathies; anemia; autoimmune hemolytic anemia; disorders of red blood cells; iron overload and iron chelation; chronic transfusion therapy; hereditary spherocytosis (HS); hereditary xerocytosis (HX); immune thrombocytopenia (ITP).
Services and Specialties
Cancer and Blood Diseases, Sickle Cell and Hemoglobin Disorders, Cerebrovascular
Interests
Identifying the causes of the cardiomyopathy and nephropathy of sickle cell disease and identifying new treatments for these complications; direct measurement of red blood cell turnover (rate of hemolysis); MRI methods for the quantitation of tissue iron; new agents for immune thrombocytopenia (ITP).
Research Areas
Hematology, Cancer and Blood Diseases
Insurance Information
Cincinnati Children's strives to accept a wide variety of health plans. Please contact your health insurance carrier to verify coverage for your specific benefit plan.
Publications
Diastolic dysfunction is associated with exercise impairment in patients with sickle cell anemia. Pediatric Blood and Cancer. 2018; 65:e27113.
Association between diffuse myocardial fibrosis and diastolic dysfunction in sickle cell anemia. Blood. 2017; 130:205-213.
Biochemical surrogate markers of hemolysis do not correlate with directly measured erythrocyte survival in sickle cell anemia. American Journal of Hematology. 2016; 91:1195-1201.
Sickle cell anemia mice develop a unique cardiomyopathy with restrictive physiology. Proceedings of the National Academy of Sciences of USA. 2016; 113:E5182-E5191.
Cardiomyopathy With Restrictive Physiology in Sickle Cell Disease. JACC-Cardiovascular Imaging. 2016; 9:243-252.
COVID-19 mRNA vaccination responses in individuals with sickle cell disease: an ASH RC Sickle Cell Research Network Study. Blood Advances. 2024; 8:4549-4553.
A pharmacokinetic-pharmacodynamic analysis of l-glutamine for the treatment of sickle cell disease: Implications for understanding the mechanism of action and evaluating response to therapy. British Journal of Haematology. 2024; 205:1147-1158.
An Institutional Approach to Equity and Improvement in Child Health Outcomes. Pediatrics. 2024; 154:e2023064994.
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