Nathan K. Evanson, MD, PhD

A photo of Nathan K. Evanson.

Attending Physician, Division of Rehabilitation Medicine

Assistant Professor, UC Department of Pediatrics

513-636-7480

Board Certified

About Me

Biography

Nathan K. Evanson, MD PhD, is originally from Southern Alberta, Canada. He obtained his bachelor's degree in biochemistry and nutritional science from Brigham Young University and went on to train in the physician scientist training program at the University of Cincinnati College of Medicine. He completed a combined residency program in pediatrics and physical medicine and rehabilitation, then joined the Division of Rehabilitation Medicine at Cincinnati Children’s. He runs the Brain Recovery After Injury (BRAIN) clinic in the division, as well as doing basic research into recovery from traumatic brain injury.

Dr. Evanson's research interests include environmental effects on neuroplasticity and recovery from brain injury, the effects of stress on recovery from brain injury, and neuroendocrine dysregulation after traumatic brain injury.

Additional Languages

Korean

Clinical Interests

Traumatic brain injury/acquired brain injury

Research Interests

Traumatic brain injury

Academic Affiliation

Assistant Professor, UC Department of Pediatrics

Divisions

Rehabilitation Medicine

My Locations

My Education

MD: University of Cincinnati, Cincinnati, OH, 2010.

PhD: University of Cincinnati, Cincinnati, OH, 2008.

Residency: Pediatrics/PM&R, University of Cincinnati/Cincinnati Children's Hospital Medical Center, Cincinnati, OH.

My Publications

Greater neurodegeneration and behavioral deficits after single closed head traumatic brain injury in adolescent versus adult male mice. Guilhaume-Correa, F; Cansler, SM; Shalosky, EM; Goodman, MD; Evanson, NK. Journal of Neuroscience Research. 2020; 98:557-570.

Connecting endoplasmic reticulum and oxidative stress to retinal degeneration, TBI, and traumatic optic neuropathy. Cansler, SM; Evanson, NK. Journal of Neuroscience Research. 2020; 98:571-574.

Early Protocolized Versus Usual Care Rehabilitation for Pediatric Neurocritical Care Patients: A Randomized Controlled Trial. Fink, EL; Beers, SR; Houtrow, AJ; Richichi, R; Burns, C; Doughty, L; Ortiz-Aguayo, R; Madurski, CA; Valenta, C; Chrisman, M; et al. Pediatric Critical Care Medicine. 2019; 20:540-550.

PICU-Based Rehabilitation and Outcomes Assessment: A Survey of Pediatric Critical Care Physicians. Treble-Barna, A; Beers, SR; Houtrow, AJ; Ortiz-Aguayo, R; Valenta, C; Chrisman, M; Smith, CM; Duffett, M; Choong, K; Watson, RS; et al. Pediatric Critical Care Medicine. 2019; 20:E274-E282.

Chronic Dysregulation of Cortical and Subcortical Metabolism After Experimental Traumatic Brain Injury. McGuire, JL; DePasquale, EA K; Watanabe, M; Anwar, F; Ngwenya, LB; Atluri, G; Romick-Rosendale, LE; McCullumsmith, RE; Evanson, NK. Molecular Neurobiology. 2019; 56:2908-2921.

Indirect traumatic optic neuropathy: modeling optic nerve injury in the context of closed head trauma. Burke, EG; Cansler, SM; Evanson, NK. Neural Regeneration Research. 2019; 14:593-594.

Membrane-initiated nuclear trafficking of the glucocorticoid receptor in hypothalamic neurons. Rainville, JR; Weiss, GL; Evanson, N; Herman, JP; Vasudevan, N; Tasker, JG. Steroids. 2019; 142:55-64.

Optic tract injury after closed head traumatic brain injury in mice: A model of indirect traumatic optic neuropathy. Evanson, NK; Guilhaume-Correa, F; Herman, JP; Goodman, MD. PLoS ONE. 2018; 13:e0197346-e0197346.

A Narrative Review of Pharmacologic and Non-pharmacologic Interventions for Disorders of Consciousness Following Brain Injury in the Pediatric Population. Evanson, NK; Paulson, AL; Kurowski, BG. Current Physical Medicine and Rehabilitation Reports. 2016; 4:56-70.

Metabotropic glutamate receptor-mediated signaling dampens the HPA axis response to restraint stress. Evanson, NK; Herman, JP. Physiology and Behavior. 2015; 150:2-7.